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224 Concussion Blood Test – Beyond MRI or CT – Lewis

concussion, blood test, TBI, CTE, recovery, prevention

Concussion blood testing is supported by the FDA & significantly contributes to the evolution of identification & care of TBI CTE – listen to these details

The post 224 Concussion Blood Test – Beyond MRI or CT – Lewis appeared first on CoreBrain Journal.

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  1. Emily on 05/29/2018 at 4:03 PM

    I am wondering if the Banyan concussion test would be useful as a potential biomarker for patients with ME/CFS and/or fibromyalgia as well? Given that several studies have shown activated microglia in these conditions, it seems like there might be increased levels of GFAP protein in this population as well, but I’m not sure if it applies to chronic illness, or just to acute injuries?

    Also, I’m curious if you see low or high levels of IgG when you test? Do you see any pattern in subclass deficiencies as well, or do you only test total IgG?

    I’d like to know if Dr Lewis megadoses the raw CBD oil at first like he does the Omega 3s? Or is it typically just one 5 mg softgel/day?

    Finally, I’d like to know if either of you have any thoughts about Coromega as a fish oil replacement. It’s much more palatable, especially for younger clients, but I wonder about the formulation. It is supposed to be more bioavailable, but that means it is also more highly processed. That said, it doesn’t seem to suffer from rancidity problems either. Curious about your opinion on this product, if you have one.

    Thank you! Great episode!

    • Dr Charles Parker on 06/03/2018 at 5:38 AM

      Thanks, Emily – I do strongly recommend this interesting article in Nature shared in your link here and briefly abstracted here:

      “Reactive astrocytes are strongly induced by central nervous system (CNS) injury and disease, but their role is poorly understood. Here we show that a subtype of reactive astrocytes, which we termed A1, is induced by classically activated neuroinflammatory microglia. We show that activated microglia induce A1 astrocytes by secreting Il-1α, TNF and C1q, and that these cytokines together are necessary and sufficient to induce A1 astrocytes. A1 astrocytes lose the ability to promote neuronal survival, outgrowth, synaptogenesis and phagocytosis, and induce the death of neurons and oligodendrocytes. Death of axotomized CNS neurons in vivo is prevented when the formation of A1 astrocytes is blocked. Finally, we show that A1 astrocytes are abundant in various human neurodegenerative diseases including Alzheimer’s, Huntington’s and Parkinson’s disease, amyotrophic lateral sclerosis and multiple sclerosis.”

      I personally do not do quantitative IgG, even though that’s an excellent question, and will ask Dr. Lewis to weigh in on your several points. My treatment experience is directed to the more frequently overlooked and disparaged qualitative IgG encouraged by such luminaries as Dr. Fasano at Harvard – his book: – Videos on IgG qualitative can be found at 1. on this PDF: – I’ll send of to Dr. Lewis this AM, thanks!

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